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By Abraham Fisher, Israel Hanin, Werner Poewe, Manfred Windisch

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Read or Download Alzheimer's and Parkinson's Diseases: Progress and New Perspectives: 8th International Conference Ad pd, Salzburg, March 2007: Abstracts (Neuro-Degenerative Diseases) PDF

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Additional info for Alzheimer's and Parkinson's Diseases: Progress and New Perspectives: 8th International Conference Ad pd, Salzburg, March 2007: Abstracts (Neuro-Degenerative Diseases)

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These patients frequently develop motor, psychiatric, and sleep-related disturbances in addition to the dementia syndrome, and information and re-assuring of patients and caregivers are important. In addition, drug treatment of patients with DLB is different from that of AD patients. DLB patients may respond better to cholinergic and dopaminergic agents, but are more likely to develop severe side-effects when treated with anti-psychotic agents, including the atypical ones. However, diagnosing DLB may be difficult.

1145delC). The effect of this mutation is to cause a frameshift (Thr382SerfsX30), creation of a premature termination codon, likely creating a null allele through nonsense mediated decay. Conclusions: These findings support the observations that 1) the corticobasal syndrome is not specific for corticobasal degeneration, 2) variable FTD/CBS features can occur in individuals with presumably the same neurodegenerative disorder, 3) familial FTD/CBS with underlying FTLD-U NII pathology can be associated with mutations in PGRN.

154delA). This mutation caused a frameshift ____________________________________ __________________________________ Neurodegenerative Dis 2007;4(suppl 1):1-350 Abstracts (Thr52HisfsX2), creating a premature termination codon, and likely created a null allele through nonsense mediated decay. 154delA mutation in PGRN. The earlier age of onset in G2 and lack of overlap in ranges of age of onset between generations raise the possibility of anticipation-like effect. J. F. M. Kuntz , M. E. W. R. M. Pickering-Brown , 3 1 1,2, R.

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